|
Many thanks to "Sleepless in Seattle" for suggesting this. The
blacker, the rarer this should be. The whiter, the more probable.
| Defect found in general population: |
|---|
| Infection found with CFIDS |
Antithrombin III
(0.2%) |
Protein-S
(0.5%) |
Protein-C
(0.8%) |
Factor V
Lieden (4%) |
Prothrombin 20210A
(2%) |
Other
(2.5%) |
Mycoplasma
(
60%) |
|
|
|
|
In Family |
|
Epstein Barr [EBV]
(57%) |
|
In Family |
|
|
|
|
Q-Fever
(
50% ) |
|
|
|
|
|
|
Rickettsia
(? 40% ?) |
|
|
|
|
|
|
Helicobacter pylori
(
37%) |
|
In Family |
|
|
|
|
Herpes [HHV-6]
(30.5%
) |
|
|
|
|
|
|
Chlamydia
(7.5%) |
|
In Family |
|
|
In Family |
|
| Est% of CFIDS |
1.8% |
4.5% |
7.2% |
36% |
18% |
21.5% |
- Many infections have many different species -- each species has resistance to
certain antibiotics.
- Multiple infections (which increases with time) is reported.
- Other illnesses are typically diagnosis as what they are (Legionnaire's
Disease, Leprosy, Tuberculosis, Syphilis, etc).
- Prior to 1993, only the first 3 hypercoagulation defects were known.
Overall 10% of the population has a hypercoagulation defect but only 75% of the defects
can currently be identified. See
Clinical Evaluation of Hypercoagulable States
| |
Home
Original 2001 WebSite as PDF for download
|
